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Sprouty homolog 3

antagonist of FGF signaling, SPRY3, HSPRY3
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Top mentioned proteins: Sprouty, Spry2, Spry4, CAN, PAR-2
Papers on antagonist of FGF signaling
Regulation of SPRY3 by X chromosome and PAR2-linked promoters in an autism susceptibility region.
Moore et al., Cork, Ireland. In Hum Mol Genet, Oct 2015
Unlike mouse Spry3, which is X-linked, human SPRY3 maps to the pseudoautosomal region 2; however, the human Y-linked allele is not expressed due to epigenetic silencing by an unknown mechanism.
Clinical and cytogenomic studies in a case of infertility associated with a nonmosaic dicentric Y chromosome.
Xia et al., Nanjing, China. In Andrologia, May 2015
array (PLCXD1→SHOX) ×1,(SRY →GOLGA2P3Y)×2, (DHRSX→ ASMT, SPRY3 →IL9R)×3.
BMP signal attenuates FGF pathway in anteroposterior neural patterning.
Han et al., South Korea. In Biochem Biophys Res Commun, 2013
Here, we show that BMP signal acts as an antagonist of FGF signaling for AP neural patterning in Xenopus embryo.
Differential actions of fibroblast growth factors on intracellular pathways and target gene expression in bovine ovarian granulosa cells.
Price et al., China. In Reproduction, 2012
Both FGF1 and FGF4 decreased levels of mRNA encoding SPRY3 and the pro-apoptotic factor BAX. FGF1 but not FGF4 stimulated expression of the cell cycle regulator, GADD45B.
Y chromosome gene expression in the blood of male patients with ischemic stroke compared with male controls.
Sharp et al., Sacramento, United States. In Gend Med, 2012
Five of these genes (VAMP7, CSF2RA, SPRY3, DHRSX, and PLCXD1) are located on pseudoautosomal regions of the human Y chromosome.
Differential expression of sprouty genes in hepatocellular carcinoma.
Tongkham et al., Bangkok, Thailand. In J Surg Oncol, 2012
METHODS: The expressions of SPRY1, SPRY2, SPRY3, and SPRY4, at the mRNA levels were determined by quantitative RT-PCR in paired HCC and non-tumor liver tissues from 31 patients.
High accuracy mutation detection in leukemia on a selected panel of cancer genes.
Aerts et al., Leuven, Belgium. In Plos One, 2011
Finally, we re-sequenced a small set of 39 candidate genes and identified recurrent mutations in TET1, SPRY3 and SPRY4.
Refinement of the X-linked nonsyndromic high-grade myopia locus MYP1 on Xq28 and exclusion of 13 known positional candidate genes by direct sequencing.
Radhakrishna et al., Omaha, United States. In Invest Ophthalmol Vis Sci, 2011
Mutation search in exons and splice junctions of candidate genes CTAG2, GAB3, MPP1, F8Bver, FUNDC2, VBP1, RAB39B, CLIC2, TMLHE, SYBL, IL9R, SPRY3, and CXYorf1 did not detect a pathogenic or predisposing variant.
Fibroblast growth factor-2 regulation of Sprouty and NR4A genes in bovine ovarian granulosa cells.
Price et al., China. In J Cell Physiol, 2011
FGF2 increased abundance of mRNA encoding SPRY1, 2, and 4, but not SPRY3.
Sprouty1 inhibits angiogenesis in association with up-regulation of p21 and p27.
Hall et al., Minneapolis, United States. In Mol Cell Biochem, 2010
Sprouty1 (Spry1) is a conserved antagonist of FGF signaling.
Revitalization of a diastemal tooth primordium in Spry2 null mice results from increased proliferation and decreased apoptosis.
Klein et al., Praha, Czech Republic. In J Exp Zool B Mol Dev Evol, 2009
We have previously shown that the arrest of a rudimentary tooth bud can be rescued by inactivating Spry2, an antagonist of FGF signaling.
[Construction and characterization of hSef recombinant adenoviral vectors].
Chang et al., Beijing, China. In Sheng Wu Gong Cheng Xue Bao, 2008
Sef (similar expression to fgf genes) was identified as a feedback antagonist of FGF signaling in zerbrafish, mouse and human.
Sprouty2 regulates growth and differentiation of human neuroblastoma cells through RET tyrosine kinase.
Takahashi et al., Nagoya, Japan. In Cancer Sci, 2007
Expression of SPRY1, SPRY2, SPRY3 and SPRY4 in HEK293T cells transfected with RET and GDNF receptor family alpha1 (GFRalpha1) genes significantly reduced sustained ERK activation as well as ELK-1 activation.
A functional interaction between sprouty proteins and caveolin-1.
Christofori et al., Basel, Switzerland. In J Biol Chem, 2006
the Sprouty/Caveolin-1 interaction modulates signaling in a growth factor- and Sprouty isoform-specific manner
Maintenance of X- and Y-inactivation of the pseudoautosomal (PAR2) gene SPRY3 is independent from DNA methylation and associated to multiple layers of epigenetic modifications.
D'Esposito et al., Napoli, Italy. In Hum Mol Genet, 2006
Recently, we demonstrated that DNA methylation plays an essential role for the maintenance of X- and Y-inactivation of the PAR2 gene SYBL1; here we report that the silencing of the second repressed PAR2 gene, SPRY3, appears to be independent of DNA methylation.
Human placental Hofbauer cells express sprouty proteins: a possible modulating mechanism of villous branching.
Yagel et al., Jerusalem, Israel. In Placenta, 2005
Spry 1, 2 and 3 expression was observed in placental tissue from all three trimesters
sprouty encodes a novel antagonist of FGF signaling that patterns apical branching of the Drosophila airways.
Krasnow et al., Stanford, United States. In Cell, 1998
Here, we describe an antagonist of FGF signaling that patterns apical branching of the Drosophila airways.
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