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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 19 Aug 2016.

Ankyrin repeat domain 28

ANKRD28, Ankyrin repeat domain 28, PITK
Top mentioned proteins: OUT, CAN, Crk, fibrillin-1, SET
Papers on ANKRD28
Polygenic inheritance of cryptorchidism susceptibility in the LE/orl rat.
Robbins et al., Wilmington, United States. In Mol Hum Reprod, Jan 2016
Associated loci contain potentially functional minor alleles (0.25-0.36 in tested rat strains) including an exonic deletion in Syne2, a large intronic insertion in Ncoa4 (an AR coactivator) and potentially deleterious variants in Solh/Capn15, Ankrd28, and Hsd17b2.
Analysis of gene profiles in glioma cells identifies potential genes, miRNAs, and target sites of migratory cells.
Chen et al., Shanghai, China. In Tumori, Sep 2015
By integrating the regulatory relationships among transcription factors, miRNAs and differentially expressed genes, we found that 7 differentially expressed genes, including SOX4, ANKRD28 and CCND1, might play crucial roles in the migration of glioma cells.
Primary urethral clear-cell adenocarcinoma: comprehensive analysis by surgical pathology, cytopathology, and next-generation sequencing.
Chinnaiyan et al., Guangzhou, China. In Am J Pathol, 2014
Transcriptome analysis revealed novel gene fusion candidates, including ANKRD28-FNDC3B.
Detection of significant pathways in osteoporosis based on graph clustering.
Xue et al., Shanghai, China. In Mol Med Report, 2012
This study provides a number of candidate genes that warrant further investigation, including DDX60, HERC5, RSAD2, SIGLEC1, CMPK2, MX1, SEPING1, EPSTI1, C9orf72, PHLDA2, PFKFB3, PLEKHG2, ANKRD28, IL1RN and RNF19B.
Genetic and epigenetic analysis of non-small cell lung cancer with NotI-microarrays.
Zabarovsky et al., Stockholm, Sweden. In Epigenetics, 2012
A comprehensive statistical analysis suggested the set of 19 gene markers, ANKRD28, BHLHE40, CGGBP1, RBSP3, EPHB1, FGD5, FOXP1, GORASP1/TTC21, IQSEC1, ITGA9, LOC285375, LRRC3B, LRRN1, MITF, NKIRAS1/RPL15, TRH, UBE2E2, VHL, WNT7A, to allow early detection, tumor progression, metastases and to discriminate between SCC and ADC with sensitivity and specificity of 80-100%.
Gene expression profiling in the leukemic stem cell-enriched CD34+ fraction identifies target genes that predict prognosis in normal karyotype AML.
Schuringa et al., Groningen, Netherlands. In Leukemia, 2011
Interestingly, for three of these genes, that is, ankyrin repeat domain 28 (ANKRD28), guanine nucleotide binding protein, alpha 15 (GNA15) and UDP-glucose pyrophosphorylase 2 (UGP2), a high transcript level was associated with a significant poorer overall survival (OS) in two independent cohorts (n=163 and n=218) of normal karyotype AML.
Regulation of focal adhesion and cell migration by ANKRD28-DOCK180 interaction.
Matsuda et al., Kyoto, Japan. In Cell Adh Migr, 2009
Recently, we searched for DOCK180-binding proteins with a nano-LC/MS/MS system, and found that ANKRD28, a protein with twenty-six ankyrin domain-repeats, interacts with the SH3 domain of DOCK180.
Ankyrin repeat domain 28 (ANKRD28), a novel binding partner of DOCK180, promotes cell migration by regulating focal adhesion formation.
Matsuda et al., Kyoto, Japan. In Exp Cell Res, 2009
ANKRD28, a novel binding partner of DOCK180, promotes cell migration by regulating focal adhesion formation.
Protein phosphatase 6 regulatory subunits composed of ankyrin repeat domains.
Brautigan et al., Charlottesville, United States. In Biochemistry, 2008
Mass spectrometry identified coprecipitating proteins as PP6 catalytic subunit and three ankyrin repeat proteins (Ankrd28, Ankrd44, and Ankrd52).
A novel gene, ANKRD28 on 3p25, is fused with NUP98 on 11p15 in a cryptic 3-way translocation of t(3;5;11)(p25;q35;p15) in an adult patient with myelodysplastic syndrome/acute myelogenous leukemia.
Bessho et al., Saitama, Japan. In Int J Hematol, 2007
We identified a novel gene fusion of ANKRD28 (ankyrin repeat domain 28) on 3p25 to NUP98 on 11p15 in a patient with adult myelodysplastic syndrome/acute myelogenous leukemia.
Dual kinase-mediated regulation of PITK by CaMKII and GSK3.
Haystead et al., Durham, United States. In Cell Signal, 2007
CaMKII and GSK3 mediate regulation of PITK.
PITK, a PP1 targeting subunit that modulates the phosphorylation of the transcriptional regulator hnRNP K.
Haystead et al., Durham, United States. In Cell Signal, 2006
These findings provide a putative mechanism by which transcriptional activity of hnRNP K can be discretely controlled through the regulation of PP1 activity.
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