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GoPubMed Proteins lists recent and important papers and reviews for proteins. Page last changed on 18 Nov 2014.

Ankyrin repeat and sterile alpha motif domain containing 1B

This gene encodes a multi-domain protein that is predominantly expressed in brain and testis. This protein interacts with amyloid beta protein precursor (AbetaPP) and may have a role in normal brain development, and in the pathogenesis of Alzheimer's disease. Expression of this gene has been shown to be elevated in patients with pre-B cell acute lymphocytic leukemia associated with t(1;19) translocation. Alternatively spliced transcript variants encoding different isoforms (some with different subcellular localization, PMID:15004329) have been described for this gene. [provided by RefSeq, Aug 2011] (from NCBI)
Top mentioned proteins: ACID, CAN, mGluR, HAD, mGluR1
Papers using AIDA antibodies
Neuroprotective activity of selective mGlu1 and mGlu5 antagonists in vitro and in vivo.
Dryer Stuart E., In PLoS ONE, 2006
... Hyclone (USA); U0126 (MAPK-ERK kinase (MEK) inhibitor), DHPG (a group I mGlus agonist); and (RS)-1-aminoindan-1,5-dicarboxylic acid (AIDA, a group I mGlus antagonist) were from Tocris Biosciences (UK); Z-DEVD-FMK (an ...
Evaluation of an improved method for mass-rearing of thrips and a thrips parasitoid
Arimura Gen-ichiro et al., In Scientific Reports, 2000
Torenia fournieri (torenia) as a model plant for transgenic studies ...
Papers on AIDA
Negative prognostic value of CD34 antigen also if expressed on a small population of acute promyelocitic leukemia cells.
Foà et al., Roma, Italy. In Ann Hematol, 30 Nov 2014
We hereby analyzed the clinico-biological features and treatment outcome of APL patients in relation to CD34 expression, even when expressed in a small subpopulation: 114 APL patients homogeneously treated with the AIDA schedule were included in the study and prognostic correlation with respect to CD34 expression, both when expressed in association with CD2 and as isolated expression (cutoff ≥2 to <10 % or ≥10 %), were investigated.
A structural mechanism for bacterial autotransporter glycosylation by a dodecameric heptosyltransferase family.
Shao et al., Beijing, China. In Elife, 13 Nov 2014
UNLABELLED: A large group of bacterial virulence autotransporters including AIDA-I from diffusely adhering E. coli (DAEC) and TibA from enterotoxigenic E. coli (ETEC) require hyper-glycosylation for functioning.
Structure and mechanism of the unique C2 domain of Aida.
Wu et al., Beijing, China. In Febs J, 31 Oct 2014
UNLABELLED: Axin interactor, dorsalization-associated (Aida) was identified as a regulatory factor that utilizes its C-terminal region to interact with axis formation inhibitor (Axin).
A molecular tool for detection and tracking of a potential indigenous Beauveria bassiana strain for managing emerald ash borer populations in Canada.
Kyei-Poku et al., Canada. In J Invertebr Pathol, 31 Oct 2014
A single nucleotide polymorphism (SNP) site, T→C was identified only in L49-1AA and was used to develop a simplified allele discrimination polymerase chain reaction (PCR) assay based on a modified allelic inhibition of displacement activity (AIDA) approach for distinguishing B. bassiana L49-1AA from all background Beauveria isolates.
An iron-containing dodecameric heptosyltransferase family modifies bacterial autotransporters in pathogenesis.
Shao et al., Beijing, China. In Cell Host Microbe, 10 Oct 2014
Although passenger domains are diverse, those found in enteric bacteria autotransporters, including AIDA-I in diffusely adhering Escherichia coli (DAEC) and TibA in enterotoxigenic E. coli, are commonly glycosylated.
Proteomics profiling of keratocystic odontogenic tumours reveals AIDA as novel biomarker candidate.
Kraljević Pavelić et al., Zagreb, Croatia. In J Oral Pathol Med, Aug 2014
CONCLUSIONS: Immunohistochemical validation of chosen putative biomarkers revealed axin interaction partner and dorsalization-antagonist (AIDA), known as a protein that blocks activation of JNK signalling pathway, as a differential biomarker for KCOT lesions on an independent cohort of KCOT tissue samples in comparison with most prevalent intra-oseal lesions inflammatory odontogenic cysts.
Crystal structure of the transport unit of the autotransporter adhesin involved in diffuse adherence from Escherichia coli.
Schmitt et al., Düsseldorf, Germany. In J Struct Biol, Jul 2014
The monomeric autotransporter AIDA-I (adhesin involved in diffuse adherence) represents an important virulence factor of these strains and is involved in adhesion, biofilm formation, aggregation and invasion into host cells.
AIDA: ab initio domain assembly server.
Godzik et al., Los Angeles, United States. In Nucleic Acids Res, Jul 2014
AIDA: ab initio domain assembly server, available at is a tool that can identify domains in multi-domain proteins and then predict their 3D structures and relative spatial arrangements.
STb and AIDA-I: the missing link?
Dubreuil, Saint-Hyacinthe, Canada. In Crit Rev Microbiol, 2010
With the recent recognition of a new adhesin, originally found in human E. coli isolates, named AIDA (adhesin involved in diffuse adherence) and its association with new E. coli pathotypes to which STb is linked, new light was shed on STb toxic potency.
The genetic background difference between diabetic patients with and without nephropathy in a Taiwanese population by linkage disequilibrium mapping using 382 autosomal STR markers.
Wu et al., Taipei, Taiwan. In Genet Test Mol Biomarkers, 2010
It is clear that some single-nucleotide polymorphisms and haplotypes of the EB-1 gene are associated with genetic difference between diabetic patients with and without nephropathy.
[Survey of recent clinical trials of the prevention and immunointervention of type 1 diabetes mellitus].
Ziegler et al., München, Germany. In Dtsch Med Wochenschr, 2010
(4) The AIDA study will test the beta-cell protective effect of interleukin-1-receptor antagonist Anakinra in 80 patients with T1DM, which has recently been shown to improve beta-cell function in patients with type 2 diabetes.
A nuclear localization signal at the SAM-SAM domain interface of AIDA-1 suggests a requirement for domain uncoupling prior to nuclear import.
Donaldson et al., Toronto, Canada. In J Mol Biol, 2009
The basic nuclear import signal for AIDA-1 is buried at the interface between the two sterile alpha motif domains.
EB1 recognizes the nucleotide state of tubulin in the microtubule lattice.
Howard et al., Dresden, Germany. In Plos One, 2008
EB1 recognizes the nucleotide state of tubulin in the microtubule lattice
The autodisplay story, from discovery to biotechnical and biomedical applications.
Meyer et al., Berlin, Germany. In Microbiol Mol Biol Rev, 2007
Various other autotransporter proteins have since been described, and the autodisplay system was developed on the basis of the natural Escherichia coli autotransporter protein AIDA-I (adhesin involved in diffuse adherence).
A beta-catenin-independent dorsalization pathway activated by Axin/JNK signaling and antagonized by aida.
Lin et al., Xiamen, China. In Dev Cell, 2007
A dorsalization pathway that is exerted by Axin/JNK signaling and its inhibitor Aida during vertebrate embryogenesis, is defined.
An influence of ligands of metabotropic glutamate receptor subtypes on parkinsonian-like symptoms and the striatopallidal pathway in rats.
Pilc et al., Kraków, Poland. In Amino Acids, 2007
Systemic or intrastriatal administration of group I mGluR antagonists (mGluR5 - MPEP, MTEP; mGluR1 - AIDA) was found to inhibit parkinsonian-like symptoms (catalepsy, muscle rigidity) in rats.
Self-associating autotransporters, SAATs: functional and structural similarities.
Sherlock et al., Denmark. In Int J Med Microbiol, 2006
In Escherichia coli, a subgroup of autotransporter proteins consists of the TibA adhesin/invasin associated with some enterotoxigenic E. coli, the AIDA adhesin from diarrhea-causing E. coli and finally, the Ag43 autoaggregation factor found in the majority of E. coli strains.
The effects of siRNA-mediated inhibition of E2A-PBX1 on EB-1 and Wnt16b expression in the 697 pre-B leukemia cell line.
Basso et al., Padova, Italy. In Haematologica, 2006
Targeted-E2A-PBX1 inhibition leads to reduced expression of the EB-1 and Wnt16b genes; aberrant expression of these genes may be a key step in leukemogenesis in t(1;19)-positive pre-B leukemia.
Extramedullary involvement at relapse in acute promyelocytic leukemia patients treated or not with all-trans retinoic acid: a report by the Gruppo Italiano Malattie Ematologiche dell'Adulto.
Mandelli et al., Bari, Italy. In J Clin Oncol, 2001
The studies investigated chemotherapy alone (LAP0389) versus RA plus chemotherapy (AIDA).
CD56 expression is an indicator of poor clinical outcome in patients with acute promyelocytic leukemia treated with simultaneous all-trans-retinoic acid and chemotherapy.
Lo Coco et al., Napoli, Italy. In J Clin Oncol, 2000
PATIENTS AND METHODS: Clinicobiologic presenting features and therapeutic results were analyzed in a series of 100 patients with genetically proven APL who were treated, according to the example of the Gruppo Italiano Malattie Ematologiche Maligne dell'Adulto multicenter trial, with ATRA plus idarubicin (AIDA) and for whom data on CD56 expression were available at diagnosis.
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