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Ankyrin repeat and sterile alpha motif domain containing 1B

This gene encodes a multi-domain protein that is predominantly expressed in brain and testis. This protein interacts with amyloid beta protein precursor (AbetaPP) and may have a role in normal brain development, and in the pathogenesis of Alzheimer's disease. Expression of this gene has been shown to be elevated in patients with pre-B cell acute lymphocytic leukemia associated with t(1;19) translocation. Alternatively spliced transcript variants encoding different isoforms (some with different subcellular localization, PMID:15004329) have been described for this gene. [provided by RefSeq, Aug 2011] (from NCBI)
Top mentioned proteins: ACID, mGluR, CAN, HAD, mGluR1
Papers using AIDA antibodies
Neuroprotective activity of selective mGlu1 and mGlu5 antagonists in vitro and in vivo.
Dryer Stuart E., In PLoS ONE, 2006
... Hyclone (USA); U0126 (MAPK-ERK kinase (MEK) inhibitor), DHPG (a group I mGlus agonist); and (RS)-1-aminoindan-1,5-dicarboxylic acid (AIDA, a group I mGlus antagonist) were from Tocris Biosciences (UK); Z-DEVD-FMK (an ...
Evaluation of an improved method for mass-rearing of thrips and a thrips parasitoid
Arimura Gen-ichiro et al., In Scientific Reports, 2000
Torenia fournieri (torenia) as a model plant for transgenic studies ...
Papers on AIDA
Comparative proteome analysis of the extracellular proteins of two Haemophilus parasuis strains Nagasaki and SW114.
Yan et al., China. In Biochem Biophys Res Commun, May 2014
The ten proteins unique to Nagasaki were putative adhesin AidA protein, putative extracellular serine protease (autotransporter) (771aa), putative extracellular serine protease (autotransporter) (780aa), protective surface antigen D15, 30S ribosomal protein S2, periplasmic serine protease do/hhoA-like protein, acid phosphatase, membrane protein, protein-disulfide isomerase, and iron ABC transporter substrate-binding protein.
Surface expression of ω-transaminase in Escherichia coli.
Larsson et al., Stockholm, Sweden. In Appl Environ Microbiol, Apr 2014
In this work, we produced an Arthrobacter citreus ω-transaminase in Escherichia coli using a surface display vector based on the autotransporter adhesin involved in diffuse adherence (AIDA-I), which has previously been used for display of dimeric proteins.
Interaction of DHPG-LTD and synaptic-LTD at senescent CA3-CA1 hippocampal synapses.
Foster et al., Gainesville, United States. In Hippocampus, Apr 2014
Application of individual antagonist for the group I mGluR (AIDA), the N-methyl-d-aspartate receptor (NMDAR) (AP-5), or L-type voltage-dependent Ca(2+) channel (VDCC) (nifedipine) failed to block synaptic-LTD and any two antagonists severely impaired synaptic-LTD induction, indicating that activation of any two mechanisms is sufficient to induce synaptic-LTD in aged animals.
A temporal examination of the planktonic and biofilm proteome of whole cell Pseudomonas aeruginosa PAO1 using quantitative mass spectrometry.
Khursigara et al., Guelph, Canada. In Mol Cell Proteomics, Apr 2014
Conversely, biofilm samples contained an uncharacterized protein with sequence similarity to an adhesion protein with self-association characteristics (AidA).
Aberrant phenotypic expression of CD15 and CD56 identifies poor prognostic acute promyelocytic leukemia patients.
Foà et al., Roma, Italy. In Leuk Res, Feb 2014
One hundred and fourteen adult patients consecutively diagnosed with PML/RARα-positive APL and homogeneously treated with the AIDA induction schedule at a single institution were included in the study.
Frequency of virulence factors in Escherichia coli isolated from suckling pigs with diarrhoea in China.
Shi et al., Harbin, China. In Vet J, Feb 2014
A total of 381 E. coli strains, obtained from 290 faecal samples from pigs on 38 farms, were tested for fimbriae (K88, K99, 987P, F41, F18, F17), non-fimbrial adhesins (AIDA-I, paa, CS31A, eae, saa), enterotoxin (LT-I, LT-II, STa, STb, EAST1), Shiga toxin (Stx1, Stx2, Stx2e), pathogenicity islands (HPI, LEE), α-haemolysin (hlyA), afa8 gene cluster (afaD, afaE) and sepA genes by PCR.
[Hippocampus quinolinic acid modulates glutamate and NMDAR/mGluR1 in chronic unpredictable mild stress-induced depression].
An et al., Xi'an, China. In Sheng Li Xue Bao, Jan 2014
Intrahippocampal injections of QUIN, QUIN antagonist Ro61-8048, non-competitive NMDA receptor antagonist MK-801 and mGluR1 antagonist AIDA were respectively adopted by rat brain stereotaxic coordinates.
Proteomic profiling of Burkholderia cenocepacia clonal isolates with different virulence potential retrieved from a cystic fibrosis patient during chronic lung infection.
Sá-Correia et al., Lisbon, Portugal. In Plos One, 2012
Also, the content of the virulence determinant AidA was higher in the last 2 isolates.
STb and AIDA-I: the missing link?
Dubreuil, Saint-Hyacinthe, Canada. In Crit Rev Microbiol, 2010
With the recent recognition of a new adhesin, originally found in human E. coli isolates, named AIDA (adhesin involved in diffuse adherence) and its association with new E. coli pathotypes to which STb is linked, new light was shed on STb toxic potency.
The genetic background difference between diabetic patients with and without nephropathy in a Taiwanese population by linkage disequilibrium mapping using 382 autosomal STR markers.
Wu et al., Taipei, Taiwan. In Genet Test Mol Biomarkers, 2010
It is clear that some single-nucleotide polymorphisms and haplotypes of the EB-1 gene are associated with genetic difference between diabetic patients with and without nephropathy.
[Survey of recent clinical trials of the prevention and immunointervention of type 1 diabetes mellitus].
Ziegler et al., München, Germany. In Dtsch Med Wochenschr, 2010
(4) The AIDA study will test the beta-cell protective effect of interleukin-1-receptor antagonist Anakinra in 80 patients with T1DM, which has recently been shown to improve beta-cell function in patients with type 2 diabetes.
A nuclear localization signal at the SAM-SAM domain interface of AIDA-1 suggests a requirement for domain uncoupling prior to nuclear import.
Donaldson et al., Toronto, Canada. In J Mol Biol, 2009
The basic nuclear import signal for AIDA-1 is buried at the interface between the two sterile alpha motif domains.
EB1 recognizes the nucleotide state of tubulin in the microtubule lattice.
Howard et al., Dresden, Germany. In Plos One, 2008
EB1 recognizes the nucleotide state of tubulin in the microtubule lattice
The autodisplay story, from discovery to biotechnical and biomedical applications.
Meyer et al., Berlin, Germany. In Microbiol Mol Biol Rev, 2007
Various other autotransporter proteins have since been described, and the autodisplay system was developed on the basis of the natural Escherichia coli autotransporter protein AIDA-I (adhesin involved in diffuse adherence).
A beta-catenin-independent dorsalization pathway activated by Axin/JNK signaling and antagonized by aida.
Lin et al., Xiamen, China. In Dev Cell, 2007
A dorsalization pathway that is exerted by Axin/JNK signaling and its inhibitor Aida during vertebrate embryogenesis, is defined.
An influence of ligands of metabotropic glutamate receptor subtypes on parkinsonian-like symptoms and the striatopallidal pathway in rats.
Pilc et al., Kraków, Poland. In Amino Acids, 2007
Systemic or intrastriatal administration of group I mGluR antagonists (mGluR5 - MPEP, MTEP; mGluR1 - AIDA) was found to inhibit parkinsonian-like symptoms (catalepsy, muscle rigidity) in rats.
Self-associating autotransporters, SAATs: functional and structural similarities.
Sherlock et al., Denmark. In Int J Med Microbiol, 2006
In Escherichia coli, a subgroup of autotransporter proteins consists of the TibA adhesin/invasin associated with some enterotoxigenic E. coli, the AIDA adhesin from diarrhea-causing E. coli and finally, the Ag43 autoaggregation factor found in the majority of E. coli strains.
The effects of siRNA-mediated inhibition of E2A-PBX1 on EB-1 and Wnt16b expression in the 697 pre-B leukemia cell line.
Basso et al., Padova, Italy. In Haematologica, 2006
Targeted-E2A-PBX1 inhibition leads to reduced expression of the EB-1 and Wnt16b genes; aberrant expression of these genes may be a key step in leukemogenesis in t(1;19)-positive pre-B leukemia.
Extramedullary involvement at relapse in acute promyelocytic leukemia patients treated or not with all-trans retinoic acid: a report by the Gruppo Italiano Malattie Ematologiche dell'Adulto.
Mandelli et al., Bari, Italy. In J Clin Oncol, 2001
The studies investigated chemotherapy alone (LAP0389) versus RA plus chemotherapy (AIDA).
CD56 expression is an indicator of poor clinical outcome in patients with acute promyelocytic leukemia treated with simultaneous all-trans-retinoic acid and chemotherapy.
Lo Coco et al., Napoli, Italy. In J Clin Oncol, 2000
PATIENTS AND METHODS: Clinicobiologic presenting features and therapeutic results were analyzed in a series of 100 patients with genetically proven APL who were treated, according to the example of the Gruppo Italiano Malattie Ematologiche Maligne dell'Adulto multicenter trial, with ATRA plus idarubicin (AIDA) and for whom data on CD56 expression were available at diagnosis.
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