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Tumor protein p53 binding protein 1

53BP1, p53-binding protein 1
Papers using 53BP1 antibodies
A Combinational Filtering Method for Enhancing Suspicious Structures in Chest X-rays
Supplier
Anderson Rhona et al., In Genome Integrity, 2010
... Cells were then incubated with primary antibody (mouse monoclonal anti-human 53BP1 (BD Biosciences Clone 19) 1:200 in ...
Changes in the cell kinetics of pig epidermis after single doses of X rays.
Supplier
Borgmann Kerstin, In PLoS ONE, 1987
... Rabbit anti-53BP1 NB100-304A31/300Acris-antibodies ...
Papers on 53BP1
The role of ATM and 53BP1 as predictive markers in cervical cancer.
GeneRIF
van Vugt et al., Groningen, Netherlands. In Int J Cancer, 2012
survival analysis revealed that ATM inhibition, but not 53BP1 depletion, strongly radiosensitised cervical cancer cells
Vaccinia-related kinase 1 (VRK1) is an upstream nucleosomal kinase required for the assembly of 53BP1 foci in response to ionizing radiation-induced DNA damage.
GeneRIF
Lazo et al., Salamanca, Spain. In J Biol Chem, 2012
Data show that vaccinia-related kinase 1 (VRK1) in resting cells plays an important role in the formation of ionizing radiation-induced foci that assemble on the 53BP1 scaffold protein during the DNA damage response.
RNF8- and RNF168-dependent degradation of KDM4A/JMJD2A triggers 53BP1 recruitment to DNA damage sites.
GeneRIF
Richard et al., Montréal, Canada. In Embo J, 2012
The authors propose that the RNF8-dependent degradation of JMJD2A regulates DNA repair by controlling the recruitment of 53BP1 at DNA damage sites.
53BP1 deficiency combined with telomere dysfunction activates ATR-dependent DNA damage response.
GeneRIF
Blasco et al., Madrid, Spain. In J Cell Biol, 2012
deletion of 53BP1 in TRF1-deficient cells impairs the C-NHEJ repair pathway, decreasing the occurrence of chromosome-type end to end telomere fusions while inducing a persistent DNA damage signal that activates the ATR-dependent DDR and HR repair pathways
Overlapping functions between XLF repair protein and 53BP1 DNA damage response factor in end joining and lymphocyte development.
GeneRIF
Zha et al., New York City, United States. In Proc Natl Acad Sci U S A, 2012
XLF repair protein and 53BP1 DNA damage response factor have overlapping functions in end joining and lymphocyte development
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